The Endocrine System

CHAPTER SYNOPSIS

This chapter discusses the endocrine system, a communication and control system that exists in the body. It distinguishes endocrine glands from exocrine glands and differentiates between hormones and prostaglandins. The principal components of the endocrine system are identified according to their location, structure, chemical secretion, effect on body tissues, and disorders associated with malfunction. Action of hormones is discussed in conjunction with target organs involved. Regulation of secretions is described as a function of negative feedback control mechanisms

LEARNING OBJECTIVES WITH RATIONALE

The student will be able to:

1. Distinguish between endocrine and exocrine glands, and define the terms hormone and prostaglandin.

Glands that discharge their secretions into ducts are called exocrine glands. Endocrine glands are ductless glands. They secrete chemicals called hormones directly into the bloodstream.

Prostaglandins, or tissue hormones, are important and extremely powerful substances found in a wide variety of body tissues. They play an important role in communication and control of many body functions but do not meet the definition of a typical hormone. A prostaglandin is produced in a tissue and diffuses only a short distance to act on cells within that tissue. Typical hormones influence and control activities of widely separated organs; typical prostaglandins influence activities of neighboring cells.

MENU

Home

Anatomy and Physiology

Introduction

Life Chemistry

Cells and Tissues

Organ Systems of the Body

Mechanisms of Disease

Integuementary System and Body Membranes

The Skeletal System

The Muscular System

The Nervous System

The Senses

The Endocrine System

Blood

The Heart and Heart Disease

The Circulation of the Blood

The Lymphatic System and Immunity

The Respiratory System

The Digestive System

Nutrition and Metabolism

urinary system.htm

Fluid and Electrolyte Balance

Acid-Base Balance

The Reproductive System

 

State of Texas Information

Exam Information

2. Identify and locate the primary endocrine glands and list the major hormones produced by each gland.

The following is a list of the primary endocrine glands, their location, and their hormonal secretions.

Pituitary gland—lies deep in the cranial cavity, in the small depression of the sphenoid bone called the sella turcica. Secretions of the anterior lobe include growth hormone, thyroid stimulating hormone, adrenocorticotropic hormone, follicle-stimulating hormone, luteinizing hormone, melanocyte-stimulating hormone, and prolactin. Secretions of the posterior lobe include antidiuretic hormone and oxytocin

Thyroid gland—lies in the neck, just below the larynx; secretes thyroxine, triiodothyronine, and calcitonin

Parathyroid glands—four small glands found on the back of the thyroid; secrete parathyroid hormone

Adrenal glands—located over the top of each kidney. The adrenal cortex secretes mineralocorticoids, glucocorticoids, and small amounts of sex hormones. The adrenal medulla secretes epinephrine and norepinephrine

Islets of Langerhans—clumps of cells scattered among pancreatic cells; secrete glucagon and insulin

Sex glands—ovaries of the female located toward the back of the pelvic cavity; secrete estrogen and progesterone. Testes of the male located in the scrotum; secrete testosterone

Thymus—located in the mediastinum; secretes thymosin

Placenta—temporary endocrine gland formed during pregnancy; secretes chorionic gonadotropin

Pineal gland—small, cone-shaped gland that lies near the roof of the third ventricle of the brain; secretes melatonin

3. Describe the mechanisms of steroid and protein hormone action.

Steroid hormones are called anabolic steroids because they stimulate the production of protein in muscles and bones. Protein hormones serve as first messenger, delivering the messages via the bloodstream and providing communication between endocrine glands and target organs. Steroid hormones can pass intact directly through the cell membrane of the target organ cell. Once inside the cell they combine with specific receptors and then enter the nucleus to influence cell activity by acting on specific genes.

4. Explain how negative and positive feedback mechanisms regulate the secretion of endocrine hormones.

Negative feedback regulates the secretion of endocrine glands by decreasing their function in response to a stimulus. An example is when the pancreas releases insulin in response to an elevation of sugar in the blood. Blood sugar levels then decrease, which causes the endocrine cells in the pancreas to cease the production and release of insulin. Most feedback systems in the body are negative. Positive feedback usually intensifies a change rather than reversing a change. Some of the positive feedback mechanisms are destructive and disrupt the homeostasis of the body. There are times that positive feedback can be helpful, such as during labor and delivery. When labor begins, the cervix becomes distended and stimulates the synthesis of oxytocin, which stimulates the uterus to contract. As the contractions increase, the levels of oxytocin increase; thus a positive feedback cycle is established. The cycle is broken by the birth of the baby.

5. Identify the principal functions of each major endocrine hormone, and describe the conditions that may result from hyposecretion or hypersecretion.

Hormones play a vital part in our survival and quality of life. When this mechanism is disturbed, the homeostasis of the body is affected. The conditions listed in the table below are those mentioned in the textbook. They develop as a result of too much or too little hormone secretion. It should be understood that other, perhaps not so obvious, conditions may exist when hormone levels are unbalanced.

MAJOR ENDOCRINE HORMONE

FUNCTIONS

POSSIBLE CONDITIONS RESULTING FROM HYPOSECRETION OR HYPERSECRETION

Pituitary Gland

Anterior pituitary gland

Thyroid-stimulating hormone (TSH)

Controls the secretion of hormones from the thyroid gland; stimulates growth of the thyroid gland; also stimulates it to secrete thyroid hormone

Hyperthyroidism (hi-per-THI-royd-izm), or oversecretion of the thyroid hormones, dramatically increases the metabolic rate. Food material is burned by the cells at an excessive rate. Individuals with this condition tend to lose weight, have an increased appetite, and show signs of nervous irritability. Hypothyroidism (hi-po-THI-royd-izm), or undersecretion of thyroid hormones, can be caused by and result in a number of different conditions, such as painless enlargement of the thyroid gland, called simple goiter.

Hyposecretion of thyroid hormones during the formative years leads to a condition called cretinism (KREE-tin-izm). It is characterized by a low metabolic rate, retarded growth and sexual development, and frequently mental retardation. Later in life, deficient thyroid hormone secretion produces the disease called myxedema (mik-se-DEE-mah).

Adrenocorticotropic hormone (ACTH)

Stimulates growth of the adrenal cortex and stimulates it to secrete glucocorticoids (mainly cortisol)

MAJOR ENDOCRINE HORMONE

FUNCTIONS

POSSIBLE CONDITIONS RESULTING FROM HYPOSECRETION OR HYPERSECRETION

Follicle-stimulating hormone (FSH)

Initiates growth of ovarian follicles each month in the ovary and stimulates one or more follicles to develop to the stage of maturity and ovulation; FSH also stimulates estrogen secretion by developing follicles; stimulates sperm production in the male

Luteinizing hormone (LH)

Acts with FSH to stimulate estrogen secretion and follicle growth to maturity; causes ovulation; causes luteinization of the ruptured follicle and stimulates progesterone secretion by corpus luteum; causes interstitial cells in the testes to secrete testosterone in the male

Melanocyte-stimulating hormone (MSH)

Causes a rapid increase in the synthesis and spread of melanin (pigment) in the skin

Growth hormone (GH)

Stimulates an increase in the size and rate of reproduction of body cells; enhances the movement of amino acids through membranes

Hypersecretion of growth hormone during the early years of life produces a condition called gigantism (ji-GAN-tizm). The name suggests the obvious characteristics of this condition. The child grows to giant size. Hyposecretion of the growth hormone produces pituitary dwarfism (DWARF-izm).

If the anterior pituitary gland secretes too much growth hormone after the normal growth years, the disease called acromegaly (ak-ro-MEG-ah-lee) develops.

Prolactin (PRL)

Sustains milk production after birth

Posterior pituitary gland

Antidiuretic hormone (ADH)

Accelerates water reabsorption from urine in the kidney tubules into the blood, thereby decreasing urine secretion

Hyposecretion of ADH results in diabetes insipidus (di-ah-BEE-tes in-SIP-i-dus), a condition in which large volumes of urine are formed.

Oxytocin (OT)

Stimulates the pregnant uterus to contract; may initiate labor; causes glandular cells of the breasts to release milk into ducts

Thyroid Gland

Thyroxine, triiodothyronine

Increases the rate at which energy is released from carbohydrates; increases the rate of protein synthesis; accelerates growth; stimulates activity in the nervous system

Calcitonin

Decreases the blood calcium concentration by inhibiting breakdown of bone, which would release calcium into the blood

Parathyroid Glands

Parathyroid hormone (PTH)

Increases blood calcium concentration by increasing the breakdown of bone with the release of calcium into the blood

With too much blood calcium, brain cells and heart cells do not function normally; a person becomes mentally disturbed, and the heart may stop. However, with too little blood calcium, nerve cells become overactive, sometimes to such a degree that they bombard muscles with so many impulses that the muscles go into spasms.

MAJOR ENDOCRINE HORMONE

FUNCTIONS

POSSIBLE CONDITIONS RESULTING FROM HYPOSECRETION OR HYPERSECRETION

Adrenal Glands

Adrenal cortex

Glucocorticoids

Help maintain normal blood glucose concentration by increasing gluconeogenesis; play an essential part in maintaining normal blood pressure; act with epinephrine and norepinephrine to produce an anti-inflammatory effect to bring about normal recovery from inflammations of various kinds; produce anti-immunity, antiallergy effects; secretion quickly increases when the body is in a condition of stress

The medical name for hypersecretion of glucocorticoids is Cushing’s syndrome. More women than men develop Cushing’s syndrome. Its most noticeable features are the so-called “moon face” and the “buffalo hump” on the upper back that develop because of the redistribution of body fat. Individuals with Cushing’s syndrome also have elevated blood sugar levels and suffer frequent infections.

Deficiency, or hyposecretion, of glucocorticoids results in a condition called Addison’s disease. Reduced cortical hormone levels result in muscle weakness, reduced blood sugar, nausea, loss of appetite, and weight loss.

Mineralocorticoids (MCs)

Increase blood sodium and decrease body potassium concentrations by accelerating kidney tubule reabsorption of sodium and excretion of potassium

Sex hormones

Small amounts of male hormones (androgens) secreted by adrenal cortex of both sexes

Adrenal medulla

Epinephrine (adrenaline), norepinephrine

Help the body resist stress by intensifying and prolonging the effects of sympathetic stimulation; increased epinephrine secretion is the first endocrine response to stress

Pancreatic Islets

Glucagon

Secreted by alpha cells; increases the blood glucose level by accelerating liver glycogenolysis (conversion of glycogen to glucose)

Insulin

Secreted by beta cells; decreases the blood glucose by accelerating the movement of glucose out of the blood into cells, which increases glucose metabolism by cells

If the pancreatic islets secrete too much insulin, as they sometimes do when a person has a tumor of the pancreas, more glucose than usual leaves the blood to enter the cells, and blood glucose decreases.

Too much insulin in the blood produces hypoglycemia (hi-po-gli-SEE-mee-ah) (lower-than-normal blood glucose concentration). Too much growth hormone produces hyperglycemia (hi-per-gli-SEE-mee-ah) (higher than normal blood glucose concentration).

If the pancreatic islets secrete too little insulin, type I (insulin-dependent) diabetes (di-ah-BEE-tes) mellitus (mell-I-tus) results.

MAJOR ENDOCRINE HORMONE

FUNCTIONS

POSSIBLE CONDITIONS RESULTING FROM HYPOSECRETION OR HYPERSECRETION

Male Sex Glands

Testosterone

The interstitial cells of testes secrete the male hormone testosterone, which masculinizes, promotes and maintains development of accessory male organs, stimulates protein anabolism

Control of secretion—high blood level of ICSH stimulates testosterone secretion; conversely, high blood level of testosterone inhibits ICSH secretion, an example of a negative feedback control mechanism

Female Sex Glands

Estrogen, progesterone

The ovaries contain two structures that secrete hormones: the ovarian follicles and the corpus luteum; graafian follicles secrete estrogens; corpus luteum secretes estrogens and progesterone

Thymus

Thymosin

Plays an important role in the development and function of the body’s immune system

Placenta

Chorionic gonadotropin

Maintain the corpus luteum during pregnancy

Estrogen, progesterone

 

6. Define diabetes insipidus, diabetes mellitus, gigantism, goiter, cretinism, and glycosuria.

Diabetes insipidus—This is a disease caused by hyposecretion of antidiuretic hormone produced by the posterior lobe of the pituitary. It causes an individual to eliminate extremely large volumes of water each day. Symptoms include severe thirst, dehydration, and electrolyte imbalances.

Diabetes mellitus—When the islets of Langerhans secrete too little insulin, a condition called diabetes mellitus results. Glucose cannot enter into cells, causing blood glucose to increase greatly. Excess sugar is filtered out of the blood by the kidneys to be lost along with urine.

Gigantism—This condition is produced when the anterior lobe of the pituitary gland secretes excessive growth hormone during the early years of life. The child grows to giant size.

Goiter—Low dietary intake of iodine causes a painless enlargement of the thyroid gland called simple goiter. This condition was once common in areas of the United States where the iodine content of soil and water was low. The use of iodized salt has dramatically reduced the incidence of simple goiter caused by low iodine intake. In simple goiter the gland enlarges in an attempt to compensate for the lack of iodine in the diet necessary for the synthesis of thyroid hormones.

Cretinism—Hyposecretion of thyroid hormones during the formative years causes cretinism. It is characterized by a low metabolic rate, retarded growth and sexual development, and frequently mental retardation.

Glycosuria—This condition is defined as excess sugar in the urine. In the case of diabetes mellitus, it results when excess blood sugar is filtered out of the body by the kidneys to be transferred to urine.

LECTURE OUTLINE

I. MECHANISMS OF HORMONE ACTION

A. Endocrine glands secrete chemicals (hormones) into the blood (Figure 10-1)

B. Hormones perform general functions of communication and control but a slower, longer-lasting type of control than that provided by nerve impulses

C. Cells acted on by hormones are called target organ cells

D. Protein hormones (first messengers) bind to receptors on the target cell membrane, triggering second messengers to affect the cell’s activities (Figure 10-2)

E. Steroid hormones bind to receptors within the target cell nucleus and influence cell activity by acting on DNA (Figure 10-3)

II. REGULATION OF HORMONE SECRETION

A. Hormone secretion is controlled by homeostatic feedback

B. Negative feedback—mechanisms that reverse the direction of a change in a physiological system (Figure 10-4)

C. Positive feedback—(uncommon) mechanisms that amplify physiological changes

III. PROSTAGLANDINS

A. Prostaglandins (PGs) are powerful substances found in a wide variety of body tissues

B. PGs are often produced in a tissue and diffuse only a short distance to act on cells in that tissue

C. Several classes of PGs include prostaglandin A (PGA), prostaglandin E (PGE), and prostaglandin F (PGF)

D. PGs influence many body functions, including respiration, blood pressure, gastrointestinal secretions, and reproduction

IV. PITUITARY GLAND (Figure 10-5)

A. Anterior pituitary gland (adenohypophysis)

1. Names of major hormones

a. Thyroid-stimulating hormone (TSH)

b. Adrenocorticotropic hormone (ACTH)

c. Follicle-stimulating hormone (FSH)

d. Luteinizing hormone (LH)

e. Melanocyte-stimulating hormone (MSH)

f. Growth hormone (GH)

g. Prolactin (lactogenic hormone)

2. Functions of major hormones

a. TSH—stimulates growth of the thyroid gland; also stimulates it to secrete thyroid hormone

b. ACTH—stimulates growth of the adrenal cortex and stimulates it to secrete glucocorticoids (mainly cortisol)

c. FSH—initiates growth of ovarian follicles each month in the ovary and stimulates one or more follicles to develop to the stage of maturity and ovulation; FSH also stimulates estrogen secretion by developing follicles; stimulates sperm production in the male

d. LH—acts with FSH to stimulate estrogen secretion and follicle growth to maturity; causes ovulation; causes luteinization of the ruptured follicle and stimulates progesterone secretion by corpus luteum; causes interstitial cells in the testes to secrete testosterone in the male

e. MSH—causes a rapid increase in the synthesis and spread of melanin (pigment) in the skin

f. GH—stimulates growth by accelerating protein anabolism; also accelerates fat catabolism and slows glucose catabolism; by slowing glucose catabolism, tends to increase blood glucose to higher than normal level (hyperglycemia)

g. Prolactin or lactogenic hormone—stimulates breast development during pregnancy and secretion of milk after the delivery of the baby

B. Posterior pituitary gland (neurohypophysis)

1. Names of hormones

a. Antidiuretic hormone (ADH)

b. Oxytocin

2. Functions of hormones

a. ADH—accelerates water reabsorption from urine in the kidney tubules into the blood, thereby decreasing urine secretion

b. Oxytocin—stimulates the pregnant uterus to contract; may initiate labor; causes glandular cells of the breasts to release milk into ducts

V. HYPOTHALAMUS

A. Actual production of ADH and oxytocin occurs in the hypothalamus

B. After production in the hypothalamus, hormones pass along axons into the pituitary gland

C. The secretion and release of posterior pituitary hormones is controlled by nervous stimulation

D. The hypothalamus controls many body functions related to homeostasis (temperature, appetite, and thirst)

VI. THYROID GLAND (Figure 10-6)

A. Names of hormones

1. Thyroid hormone—thyroxine (T4) and triiodothyronine (T3)

2. Calcitonin

B. Functions of hormones

1. Thyroid hormones—accelerate catabolism (increase the body’s metabolic rate)

2. Calcitonin—decreases the blood calcium concentration by inhibiting breakdown of bone, which would release calcium into the blood

VII. PARATHYROID GLANDS (Figure 10-6)

A. Name of hormone—parathyroid hormone (PTH)

B. Function of hormone—increases blood calcium concentration by increasing the breakdown of bone with the release of calcium into the blood

VIII. ADRENAL GLANDS (Figure 10-9)

A. Adrenal cortex

1. Names of hormones (corticoids)

a. Glucocorticoids (GCs)—chiefly cortisol (hydrocortisone)

b. Mineralocorticoids (MCs)—chiefly aldosterone

c. Sex hormones—small amounts of male hormones (androgens) secreted by adrenal cortex of both sexes

2. Cell layers (zones)

a. Outer layer—secretes mineralocorticoids

b. Middle layer—secretes glucocorticoids

c. Inner layer—secretes sex hormones

3. Mineralocorticoids—increase blood sodium and decrease body potassium concentrations by accelerating kidney tubule reabsorption of sodium and excretion of potassium

4. Functions of glucocorticoids

a. Help maintain normal blood glucose concentration by increasing gluconeogenesis—the formation of “new” glucose from amino acids produced by the breakdown of proteins, mainly those in muscle tissue cells; also the conversion to glucose of fatty acids produced by the breakdown of fats stored in adipose tissue cells

b. Play an essential part in maintaining normal blood pressure—make it possible for epinephrine and norepinephrine to maintain a normal degree of vasoconstriction, a condition necessary for maintaining normal blood pressure

c. Act with epinephrine and norepinephrine to produce an anti-inflammatory effect, to bring about normal recovery from inflammations of various kinds

d. Produce anti-immunity, antiallergy effect; bring about a decrease in the number of lymphocytes and plasma cells and therefore a decrease in the amount of antibodies formed

e. Secretion of glucocorticoid quickly increases when the body is thrown into a condition of stress; high blood concentration of glucocorticoids, in turn, brings about many other stress responses (Figure 9-10)

B. Adrenal medulla

1. Names of hormones—epinephrine (adrenaline) and norepinephrine

2. Functions of hormones—help the body resist stress by intensifying and prolonging the effects of sympathetic stimulation; increased epinephrine secretion is the first endocrine response to stress

IX. PANCREATIC ISLETS (Figure 10-11)

A. Names of hormones

1. Glucagon—secreted by alpha cells

2. Insulin—secreted by beta cells

B. Functions of hormones

1. Glucagon increases the blood glucose level by accelerating liver glycogenolysis (conversion of glycogen to glucose)

2. Insulin decreases the blood glucose by accelerating the movement of glucose out of the blood into cells, which increases glucose metabolism by cells

X. FEMALE SEX GLANDS

The ovaries contain two structures that secrete hormones—the ovarian follicles and the corpus luteum

A. Effects of estrogen (feminizing hormone)

1. Development and maturation of breasts and external genitals

2. Development of adult female body contours

3. Initiation of menstrual cycle

XI. MALE SEX GLANDS

The interstitial cells of testes secrete the male hormone testosterone

A. Effects of testosterone (masculinizing hormone)

1. Maturation of external genitals

2. Beard growth

3. Voice changes at puberty

4. Development of musculature and body contours typical of the male

XII. THYMUS

A. Name of hormone—thymosin

B. Function of hormone—plays an important role in the development and function of the body’s immune system

XIII. PLACENTA

A. Name of hormones—chorionic gonadotropins, estrogens, and progesterone

B. Functions of hormones—maintain the corpus luteum during pregnancy

XIV. PINEAL GLAND

A. A cone-shaped gland near the roof of the third ventricle of the brain

1. Glandular tissue predominates in children and young adults

2. Becomes fibrous and calcified with age

B. Called third eye because its influence on secretory activity is related to the amount of light entering the eyes

C. Secretes melatonin, which:

1. Inhibits ovarian activity

2. Regulates the body’s internal clock

XV. OTHER ENDOCRINE STRUCTURES

A. Many organs (for example, the stomach, intestines, and kidney) produce endocrine hormones

B. The atrial wall of the heart secretes atrial natriuretic hormone (ANH), which stimulates sodium loss from the kidneys

C. Fat-storing cells secrete leptin, which controls how full or hungry we feel